- •Burn Care and Treatment
- •Contents
- •1.1 Initial Assessment and Emergency Treatment
- •Box 1.1. Primary and Secondary Survey
- •1.2 Fluid Resuscitation and Early Management
- •1.2.1 Fluid Resuscitation
- •1.2.2 Endpoint of Burn Resuscitation
- •1.2.4.2 Hypertonic Saline
- •1.2.4.3 Antioxidants: High-Dose Vitamin C
- •1.2.4 Role of Colloids, Hypertonic Saline, and Antioxidants in Resuscitation
- •1.2.4.1 Colloids
- •1.3 Evaluation and Early Management of Burn Wound
- •1.3.1 Evaluation of Burn Depth
- •1.3.2 Choice of Topical Dressings
- •1.3.3 Escharotomy
- •1.3.4 Operative Management
- •References
- •2: Pathophysiology of Burn Injury
- •2.1 Introduction
- •2.2 Local Changes
- •2.2.1 Temperature and Time Effect
- •2.2.2 Etiology
- •2.2.3 Pathophysiologic Changes
- •2.2.4 Burn Size
- •2.3 Systemic Changes
- •2.3.1 Edema Formation
- •2.3.3.1 Resting Energy Expenditure
- •2.3.3.2 Muscle Catabolism
- •2.3.3.3 Glucose and Lipid Metabolism
- •2.3.4 Renal System
- •2.3.5 Gastrointestinal System
- •2.3.6 Immune System
- •2.4 Summary and Conclusion
- •References
- •3: Wound Healing and Wound Care
- •3.1 Introduction
- •3.2 Physiological Versus Pathophysiologic Wound Healing
- •3.2.1 Transforming Growth Factor Beta
- •3.2.2 Interactions Between Keratinocytes and Fibroblasts
- •3.2.3 Matrix Metalloproteinases (MMP)
- •3.3.1 Burn Wound Excision
- •3.3.2 Burn Wound Coverage
- •3.3.3 Autografts
- •3.3.4 Epidermal Substitutes
- •3.3.5 Dermal Substitutes
- •3.3.6 Epidermal/Dermal Substitutes
- •3.4 Summary
- •References
- •4: Infections in Burns
- •4.1 Burn Wound Infections
- •4.1.1 Diagnosis and Treatment of Burn Wound Infections
- •4.1.1.1 Introduction
- •4.1.2 Common Pathogens and Diagnosis
- •4.1.3 Clinical Management
- •4.1.3.1 Local
- •4.1.3.2 Systemic
- •4.1.4 Conclusion
- •4.4 Guidelines for Sepsis Resuscitation
- •References
- •5: Acute Burn Surgery
- •5.1 Introduction
- •5.2 Burn Wound Evaluation
- •5.3 Escharotomy/Fasciotomy
- •5.4 Surgical Burn Wound Management
- •5.5.1 Face
- •5.5.2 Hands
- •5.6 Treatment Standards in Burns Larger Than Sixty Percent TBSA
- •5.7 Temporary Coverage
- •5.9.1 Early Mobilisation
- •5.9.2 Nutrition and Anabolic Agents
- •Bibliography
- •6.1 Introduction
- •6.2 Initial and Early Hospital Phase
- •6.2.1 Blood Pressure
- •6.2.1.1 Resuscitation
- •6.2.1.2 Albumin
- •6.2.1.3 Transfusion
- •6.2.1.4 Vasopressors
- •6.2.2 Urine Output
- •6.2.4 Respiration
- •6.2.4.1 Ventilation Settings
- •6.2.5 Inhalation Injury
- •6.2.6 Invasive and Noninvasive Thermodilution Catheter (PiCCO Catheter)
- •6.2.7 Serum Organ Markers
- •6.3 Later Hospital Phase
- •6.3.1 Central Nervous System
- •6.3.1.1 Intensive Care Unit-Acquired Weakness
- •6.3.1.2 Thermal Regulation
- •6.3.2 Heart
- •6.3.3 Lung
- •6.3.3.1 Ventilator-Associated Pneumonia
- •6.3.4 Liver/GI
- •6.3.4.1 GI Complications/GI Prophylaxis/Enteral Nutrition
- •6.3.4.2 Micronutrients and Antioxidants
- •6.3.5 Renal
- •6.3.6 Hormonal (Thyroid, Adrenal, Gonadal)
- •6.3.7.3 Phosphate and Magnesium
- •6.3.7.4 Calcium
- •6.3.7 Electrolyte Disorders
- •6.3.7.1 Sodium
- •6.3.7.2 Chloride
- •6.3.8 Bone Demineralization and Osteoporosis
- •6.3.9 Coagulation and Thrombosis Prophylaxis
- •Conclusion
- •References
- •7.1 Introduction
- •7.2.1 Glucose Metabolism
- •7.2.2 Fat Metabolism
- •7.2.3 Protein Metabolism
- •7.3 Attenuation of the Hypermetabolic Response
- •7.3.1.1 Nutrition
- •Nutritional Route
- •Initiation of Nutrition
- •Amount of Nutrition
- •Composition of Nutrition (Table 7.1)
- •7.3.1.2 Early Excision
- •7.3.1.3 Environmental Support
- •7.3.1.4 Exercise and Adjunctive Measures
- •7.3.2 Pharmacologic Modalities
- •7.3.2.1 Recombinant Human Growth Hormone
- •7.3.2.2 Insulin-Like Growth Factor
- •7.3.2.3 Oxandrolone
- •7.3.2.4 Propranolol
- •7.3.2.5 Insulin
- •7.3.2.6 Metformin
- •7.3.2.7 Other Options
- •7.4 Summary and Conclusion
- •References
- •8.1 Introduction
- •8.2 Knowledge Base
- •8.2.1.1 Incidence
- •8.3 Aetiology and Risk Factors
- •8.3.1 Pathophysiology
- •8.3.1.1 Severity Factors
- •Box 8.1. Burn Severity Factors
- •8.3.2 Local Damage
- •8.3.3 Fluid and Electrolyte Shifts
- •8.4 Cardiovascular, Gastrointestinal and Renal System Manifestations
- •8.4.1 Types of Burn Injuries
- •8.4.1.1 Clinical Manifestations
- •Box 8.2. Primary Survey Assessment
- •Box 8.3. Signs and Symptoms of Hypovolemic Shock
- •Box 8.4. Physical Findings of Inhalation Injury
- •Box 8.5. Signs and Symptoms of Vascular Compromise
- •Box 8.6. Secondary Survey Assessment
- •8.5 Clinical Management
- •8.5.1 Nonsurgical Care
- •Box 8.7. Secondary Survey Highlights
- •Box 8.8. First Aid Management at the Scene
- •Box 8.9. Treatment of the Severely Burned Patient on Admission
- •Box 8.10. Fluid Resuscitation Using the Parkland (Baxter) Formula
- •Box 8.11. Properties of Topical Antimicrobial Agents
- •Box 8.12. Criteria for Burn Wound Coverings
- •8.5.2 Surgical Care
- •8.5.3 Pharmacological Support
- •8.5.4 Psychosocial Support
- •References
- •9.1 Electrical Injuries
- •9.1.1 Introduction
- •9.1.2 Diagnosis and Management
- •9.2 Chemical Burns
- •9.3 Cold Injury (Frostbite)
- •References
- •10.1 Introduction
- •10.2 Pathophysiology
- •10.3 Scarring
- •10.4 Therapy
- •10.5 Psychological Aspects
- •10.6 Return to Work
- •10.8 Exercise
- •10.9 Summary
- •References
- •11: Burn Reconstruction Techniques
- •11.1 From the Reconstructive Ladder to the Reconstructive Elevator
- •11.2 The Reconstructive Clockwork
- •11.2.1 General Principles
- •11.3 Indication and Timing of Surgical Intervention
- •11.4 The Techniques of Reconstruction
- •11.4.1 Excision Techniques
- •11.4.1.1 W-Plasty and Geometric Broken Line Closure
- •11.4.2 Serial Excision and Tissue Expansion
- •11.4.3 Skin Grafting Techniques
- •11.4.4 Local Skin Flaps
- •11.4.4.1 Z-Plasty
- •11.4.4.2 Double Opposing Z-Plasty
- •11.4.4.3 ¾ Z-plasty or half-Z
- •11.4.4.4 Musculocutaneous (MC) or Fasciocutaneous (FC) Flap Technique
- •11.4.5 Distant Flaps
- •11.4.5.1 Free Tissue Transfer
- •11.4.5.2 Perforator Flaps
- •11.4.6 Composite Tissue Allotransplantation
- •11.4.7 Regeneration: Tissue Engineering
- •11.4.8 Robotics/Prosthesis
- •11.5 Summary
- •References
- •Appendix
- •Sedatives and Pain Medications
- •Index
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Healthy, granulation tissue is apparent on the clean wound bed, and re-epi- thelialising cells are seen to migrate from the wound edges and the dermal bed to slowly close the wound within 10–14 days. Full-thickness wounds have a thicker, more leathery eschar, which does not separate easily from the viable wound bed. Those wounds require surgical excision and grafting. Continuous assessment of the patient’s systemic response to the burn injury is an essential part of an individualised plan of care. Subtle changes quickly identified by the burn team can prevent complications from occurring or worsening over time.
•During the final, rehabilitative phase, attention turns to scar maturation, contracture development and functional independence issues. The areas of burn, which heal either by primary intention or skin grafting, initially appear red or pink and are flat. Layers of re-epithelialising cells continue to form, and collagen fibres in the lower scar tissue add strength to a fragile wound. Over the next month, the scars may become more red from increased blood supply and more raised from disorganised whorls of collagen and fibroblasts/ myofibroblasts. The scars are referred to as hypertrophic in nature. If oppositional forces are not applied through splinting devices, exercises or stretching routines, this new tissue continues to heal by shortening and forming contractures. A certain amount of contracture development is unavoidable, but the impact can be lessened through prompt and aggressive interventions.
8.5Clinical Management
8.5.1Nonsurgical Care
Emergent Phase Priorities: airway management, fluid therapy, initial wound care Emergent Phase Goals of Care: initial assessment, management and stabilisation of
the patient during the first 48 h post-burn
Emergent Phase Assessment
•During the rapid, primary survey, airway and breathing assume top priority. A compromised airway requires prompt attention and breath sounds verified in each lung field.
•If circumferential, full-thickness burns are present on the upper trunk and back, ventilation must be closely monitored as breathing might be impaired and releasing escharotomies necessary (Fig. 8.8).
•Spine must be stabilised until c-spines are cleared.
•Circulation is assessed by examining skin colour, sensation, peripheral pulses and capillary filling. Circumferential, full-thickness burns to the arms or legs must be assessed via palpation or Doppler for evidence of adequate circulation. Escharotomies might be required.
•Typically, burn patients are alert and oriented the first few hours post-burn. If that is not the case, consideration must be given to associated head injury
8 Nursing Management of the Burn-Injured Person |
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Fig. 8.8 Full-thickness flame burn with releasing escharotomies
(including a complete neurological assessment), substance abuse, hypoxia or pre-existing medical conditions.
•All clothing and jewellery need to be removed in order to visualise the entire body and avoid the “tourniquet-like” effect of constricting items left in place as oedema increases.
•Adherent clothing needs to be gently soaked off with normal saline to avoid further trauma and unnecessary pain.
•Prompt fluid resuscitation must be initiated to address hypovolemic shock.
•Secondary, head-to-toe survey rules out any associated injuries. All medical problems are identified and managed in a timely fashion.
•Circumstances of the injury must be explored to understand the mechanism, duration and severity of the injury.
•Patient’s pertinent medical history includes identification of pre-existing disease or associated illness (cardiac or renal disease, diabetes, hypertension), medication/alcohol/drug history, allergies and tetanus immunisation status. A handy mnemonic can be used to remember this information (Box 8.7).
Box 8.7. Secondary Survey Highlights
A |
Allergies |
M |
Medications |
P |
Previous illness, past medical history |
L |
Last meal or drink |
|
|
E |
Events preceding injury |
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126 J. Knighton
Box 8.8. First Aid Management at the Scene
Steps |
Action |
Step 1 |
Stop the burning process – remove patient from heat source |
Step 2 |
Maintain airway – resuscitation measures may be necessary |
Step 3 |
Assess for other injuries and check for any bleeding |
|
|
Step 4 |
Flush chemical burns copiously with cool water |
|
|
Step 5 |
Flush other burns with cool water to comfort |
Step 6 |
Protect wounds from further trauma |
Step 7 |
Provide emotional support and have someone to remain |
|
with patient to explain help is on the way |
Step 8 |
Transport the patient as soon as possible to nearby |
|
emergency department |
Emergent Phase Management
•The top priority of care is to stop the burning process (Box 8.8). During the initial first aid period at the scene, the patient must be removed from the heat source, chemicals should be brushed off and/or flushed from the skin, and the patient wrapped in a clean sheet and blanket ready for transport to the nearest hospital. Careful, local cooling of the burn wound with saline-moistened gauze can continue as long as the patient’s core temperature is maintained and he/she does not become hypothermic.
–Upon arrival at the hospital, the burned areas can be cooled further with normal saline, followed by a complete assessment of the patient and initiation of emergency treatment (Box 8.9). In a burn centre, the cooling may take place, using a cart shower system, in a hydrotherapy room (Fig. 8.9). The temperature of the water is adjusted to the patient’s comfort level, but tepid is usually best, while the wounds are quickly cleaned and dressings applied.
•Airway management includes administration of 100 % oxygen if burns are 20 % body surface area or greater. Suctioning and ventilatory support may be necessary. If the patient is suspected of having or has an inhalation injury, intubation needs to be performed quickly.
•Evidence-based procedures for the insertion of central lines and care of ventilated patients have resulted in impressive reductions in central line blood stream infection rates and ventilator-acquired pneumonia (VAP) [3].
•Circulatory management includes intravenous infusion of fluid to counteract the effects of hypovolemic shock for adult patients with burns >15 % body surface area and children with burns >10 % body surface area. Upon admission, two large bore, intravenous catheters should be inserted, preferably into, but not limited to, unburned tissue.
8 Nursing Management of the Burn-Injured Person |
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Box 8.9. Treatment of the Severely Burned Patient on Admission
Steps Action
Step 1 Stop the burning process
Step 2 Establish and maintain an airway; inspect face and neck for singed nasal hair, soot in the mouth or nose, stridor or hoarseness
Step 3 Administer 100 % high-flow humidified oxygen by non-rebreather mask. Be prepared to intubate if respiratory distress increases
Step 4 Establish intravenous line(s) with large bore cannula(e) and initiate fluid replacement using Lactated Ringer’s solution
Step 5 Insert an indwelling urinary catheter
Step 6 Insert a nasogastric tube
Step 7 Monitor vital signs including level of consciousness and oxygen saturation
Step 8 Assess and control pain
Step 9 Gently remove clothing and jewellery
Step 10 Examine and treat other associated injuries
Step 11 Assess extremities for pulses, especially with circumferential burns
Step 12 Determine depth and extent of the burn
Step 13 Provide initial wound care – cool the burn and cover with large, dry gauze dressings
Step 14 Prepare to transport to a burn centre as soon as possible
Fig. 8.9 Cart shower for hydrotherapy
